BlueCross and BlueShield of Montana Medical Policy/Codes
Femoroacetabular Impingement, Surgical Treatment
Chapter: Surgery: Procedures
Current Effective Date: October 25, 2013
Original Effective Date: December 18, 2009
Publish Date: October 25, 2013
Revised Dates: December 21, 2010; September 15, 2011; May 8, 2012; September 4, 2013
Description

Femoroacetabular impingement (FAI) results from localized compression in the joint due to an anatomical mismatch between the head of the femur and the acetabulum. Symptoms of impingement typically occur in young to middle-aged adults prior to the onset of osteoarthritis but may be present in younger patients with developmental hip disorders. The objective of surgical treatment of FAI is to improve symptoms and reduce further damage to the joint.

Background

FAI arises from an anatomical mismatch between the head of the femur and the acetabulum, causing compression of the labrum or articular cartilage during flexion. The mismatch can arise from subtle morphologic alterations in the anatomy or orientation of the ball-and-socket components (for example, a bony prominence at the head-neck junction or acetabular overcoverage) with articular cartilage damage initially occurring from abutment of the femoral neck against the acetabular rim, typically at the anterosuperior aspect of the acetabulum. Although hip joints can possess the morphologic features of FAI without symptoms, FAI may become pathologic with repetitive movement and/or increased force on the hip joint. High-demand activities may also result in pathologic impingement in hips with normal morphology.

Two types of impingement, known as cam impingement and pincer impingement, may occur alone or, more frequently, together. Cam impingement is associated with an asymmetric or nonspherical contour of the head or neck of the femur jamming against the acetabulum, resulting in cartilage damage and delamination (detachment from the subchondral bone). Deformity of the head/neck junction that looks like a pistol grip on radiographs is associated with damage to the anterosuperior area of the acetabulum. Symptomatic cam impingement is found most frequently in young male athletes. Pincer impingement is associated with overcoverage of the acetabulum and pinching of the labrum, with pain more typically beginning in women of middle age. In cases of isolated pincer impingement, the damage may be limited to a narrow strip of the acetabular cartilage. It has been proposed that impingement with damage to the labrum and/or acetabulum is a causative factor in the development of hip osteoarthritis and that as many as half of cases currently categorized as primary osteoarthritis may have an etiology of FAI.

Previously, access to the joint space was limited, and treatment consisted primarily of debridement and/or labral reattachment. A technique for hip dislocation with open osteochondroplasty that preserved the femoral blood supply was reported by Ganz and colleagues in 2001. Visualization of the entire joint with this procedure led to the identification and acceptance of FAI as an etiology of cartilage damage (the association between abnormal femoral head/neck morphology and early age onset of osteoarthritis had been described earlier by others) and the possibility of correcting the abnormal femoroacetabular morphology. Open osteochondroplasty of bony abnormalities and treatment of the symptomatic cartilage defect is considered the gold standard for complex bony abnormalities. However, open osteochondroplasty is invasive, requiring transection of the greater trochanter (separation of the femoral head from the femoral shaft) and dislocation of the hip joint to provide full access to the femoral head and acetabulum. In addition to the general adverse effects of open surgical procedures, open osteochondroplasty with dislocation has been associated with nonunion and neurologic and soft tissue lesions. Less-invasive hip arthroscopy and an arthroscopy-assisted mini-approach were adapted from the open approach by 2004. Arthroscopy requires specially designed instruments and is considered to be more technically difficult due to reduced visibility and limited access to the joint space. Advanced imaging techniques, including computed tomography (CT) and fluoroscopy, have been utilized to improve visualization of the 3-dimensional head/neck morphology during arthroscopy.

An association between FAI and athletic pubalgia, sometimes called sports hernia, has been proposed. Athletic pubalgia is an umbrella term for a large variety of musculoskeletal injuries involving attachments and/or soft tissue support structures of the pubis. It is believed that if FAI presents with limitations in hip range of motion, compensatory patterns during athletic activity may lead to increased stresses involving the abdominal obliques, distal rectus abdominis, pubic symphysis, and adductor musculature. The condition is more common in men than in women and is associated with sports in which high speed twisting of the hip and pelvis occur (e.g., football and hockey). Under surgical exploration, a variety of musculotendinous defects, nerve entrapments, and inflammatory conditions have been observed. These defects are often discovered and repaired during open or minimally invasive exploratory laparoscopy. Surgery for athletic pubalgia has been performed concurrently with treatment of FAI or might be performed following FAI surgery if symptoms do not resolve.

The recognition and treatment of FAI has also brought attention to the possibility of cam-type FAI after slipped capital femoral epiphysis (SCFE). The standard treatment for SCFE is stabilization across the physis by in-situ pinning, although it is not uncommon for patients with SCFE to develop premature osteoarthritis requiring total hip arthroplasty (THA) within 20 years. Treatments being evaluated for pediatric patients with SCFE-related FAI include osteoplasty without dislocation, or with the open dislocation technique described by Ganz. The Ganz technique (capital realignment with open dislocation) is technically demanding with a steep learning curve and a high risk of complications. Therefore, early treatment to decrease impingement must be weighed against increased risk for adverse events including avascular necrosis in patients with SCFE.

It is known that surgical treatment of FAI pathology is less effective for pain reduction in patients with late stage osteoarthritis. In addition, delay in the surgical correction of bony abnormalities may lead to disease progression to the point at which joint preservation is no longer appropriate. It is believed that osteoplasty of the impinging bone is needed to protect the cartilage from further damage and to preserve the natural joint. Therefore, if FAI morphology is shown to be an etiology of osteoarthritis, a future strategy to reduce the occurrence of idiopathic hip osteoarthritis could be early recognition and treatment of FAI before cartilage damage occurs.

Policy

Each benefit plan, summary plan description or contract defines which services are covered, which services are excluded, and which services are subject to dollar caps or other limitations, conditions or exclusions.  Members and their providers have the responsibility for consulting the member's benefit plan, summary plan description or contract to determine if there are any exclusions or other benefit limitations applicable to this service or supply.  If there is a discrepancy between a Medical Policy and a member's benefit plan, summary plan description or contract, the benefit plan, summary plan description or contract will govern.

Medically Necessary

Blue Cross and Blue Shield of Montana (BCBSMT) may consider open or arthroscopic surgical treatment of femoroacetabular impingement (FAI) medically necessary when ALL of the following conditions have been met:

1.  Age

  • skeletally mature with documented closure of growth plates (e.g., 15 years or older); AND

2.  Symptoms

  • Moderate-to-severe hip pain that is worsened by flexion activities (e.g., squatting or prolonged sitting) that significantly limits activities; AND 
  • Unresponsive to conservative therapy for at least three months (including activity modifications, restriction of athletic pursuits and avoidance of symptomatic motion); AND
  • Positive impingement sign on clinical examination (pain elicited with 90 degrees of flexion and internal rotation and adduction of the femur); AND

3.  Imaging

  • Morphology indicative of cam or pincer-type FAI, e.g., pistol-grip deformity, femoral head-neck offset with an alpha angle greater than 50 degrees, a positive wall sign, acetabular retroversion (over-coverage with crossover sign), coxa profunda or protrusion, or damage of the acetabular rim; AND 
  • High probability of a causal association between the FAI morphology and damage, e.g., a pistol-grip deformity with a tear of the acetabular labrum and articular cartilage damage in the anterosuperior quadrant; AND
  • No evidence of advanced osteoarthritis (OA), defined as Tonnis grade II or III, or joint space of less than 2 mm; AND 
  • No evidence of severe (Outerbridge grade IV) chondral damage.

Investigational

BCBSMT considers open or arthroscopic surgical treatment of FAI experimental, investigational and unproven in all other situations.

Policy Guidelines

There are no specific CPT codes for the open treatment of FAI. The procedures will likely be coded using unlisted CPT codes such as code 27299. Effective in 2011, there are specific CPT codes for the arthroscopic approach: 29914, 29915, 29916.

Rationale

This policy was updated with a literature search performed through April 2013. The key issue for this policy is whether correction of femoroacetabular impingement (FAI) morphology with open or arthroscopic osteoplasty alters the development of symptomatic cartilage damage and hip osteoarthritis. Given the relatively recent recognition of FAI and development of interventional procedures, neither the natural history of FAI nor the effect of osteochondroplasty on the development of osteoarthritis is known. No randomized controlled trials (RCTs) were identified. Therefore, to evaluate the potential benefit of FAI with the evidence available at this time, studies were reviewed for the following:

  • Evidence that FAI is an etiology of cartilage damage and hip osteoarthritis.
  • Evidence for benefit of open or arthroscopic osteoplasty on pain and function in patients with FAI pathology. If there is benefit, what are the specific indications and the appropriate timing for surgical intervention?

Natural History

A frequently cited paper describing the relationship between hip morphology and acetabular damage is from the group of Ganz and Leunig, who had previously reported the open procedure with dislocation in 2001. (1) In this study, a total of 26 patients with pure pistol-grip deformity and 16 patients with isolated coxa profunda were identified from 302 hips treated for intra-articular pathology between 1996 and 2001. Only hips with minor radiologic changes, with narrowing or osteophytes equivalent to an osteoarthrosis grade less than one according to the classification of Tonnis, were included. Excluded were hips with traumatic or post-traumatic conditions (n=37), avascular necrosis (n=14), and hips that had underdone previous surgery (n=7). Patients with incomplete or inadequate preoperative radiographs were also excluded. For the 26 hips that met the inclusion/exclusion criteria and showed isolated cam impingement on preoperative radiographs, all showed acetabular cartilage damage in the anterosuperior area of the acetabulum with separation between the acetabular cartilage and the labrum. In the 16 hips with isolated pincer impingement, the damage was located more circumferentially, usually including only a narrow strip of the acetabular cartilage. The report illustrated that in carefully selected patients with early stage osteoarthritis and well-defined hip configurations, a strong association exists between specific hip morphology and the pattern of cartilage damage. The intent of the study was "to obtain unequivocal data" on the starting point of joint degeneration with FAI; damage in patients with more complex morphology was not described.

Population Studies. To address the gap in current knowledge, Ganz and colleagues began a population-based natural history study in 2005 with a cohort of over 1,000 young men to determine whether morphologic alterations are associated with an increased rate of early osteoarthritis. As of 2011, 1,080 asymptomatic young men in the Sumiswald Cohort had undergone clinical examination and completed the Western Ontario and McMaster Universities Osteoarthritis Index (WOMAC) and the EuroQol health-related quality-of-life questionnaire. (2) Of these, 244 randomly selected subjects with a mean age of 19.9 years underwent magnetic resonance imaging (MRI) to evaluate cam-type deformities, labral lesions, cartilage thickness, and impingement pits. Definite cam-type deformities were detected in 67 asymptomatic men (27%). The primary outcome of labral lesions was found in a large proportion of subjects both with and without cam-type deformities; labral lesions were found 57 of 67 participants (85%) with a cam-type deformity and 118 of 177 participants (67%) without a deformity. Logistic regression models adjusted for age and body mass index (BMI) found an odds ratio [OR] of 2.77 for labral lesions, 2.91 for impingement pits, and 2.45 for labral deformities. Cartilage thickness was -0.19 mm lower in subjects with cam-type deformities compared to those without in this cross-sectional study. As noted by the authors, longitudinal studies are needed to determine whether cam-type deformity is a risk factor for symptomatic hip osteoarthritis.

In 2010, Gosvig et al. published findings from a cross-sectional radiographic and questionnaire database of 4,151 individuals from the Copenhagen Osteoarthritis study. Subjects in this population-based cohort were selected according to a random Social Security number algorithm between 1991 and 1994. (3) Excluding subjects with hip replacement surgery, Perthes disease, childhood hip disease, rheumatoid arthritis, radiographs with excessive rotation, or unreadable radiographs resulted in 3,620 subjects who met the study criteria. The study group consisted of 1,332 men with a mean age of 60.0 years (range 22 to 90 years) and 2,288 women with a mean age of 60.8 years (range 21 to 90 years). The hips were categorized as being without malformations or as having an abnormality consisting of a deep acetabular socket, a pistol-grip deformity, or a combination of the 2 on the basis of radiographic criteria. The male and female prevalence of hip-joint malformations was 71% and 36.6%, respectively. The prevalence of hip osteoarthritis, defined radiographically as a minimum joint-space width of equal to or less than 2 mm, was 9.5% in men and 11.2% in women. Although there was no significant increase in the reporting of deep groin pain in subjects with hip-joint malformations (p>0.13), a deep acetabular socket or pistol-grip deformity were significant risk factors in the development of hip osteoarthritis (risk ratio of 2.4 and 2.2, respectively).

The prevalence of a positive FAI impingement test was assessed in a population-based study of healthy 19-year-olds. (4) The study included questionnaires on medical and functional status, a clinical hip examination including the impingement test and hip range of motion, and two pelvic radiographs (anterior-posterior and frog-leg views). Out of 1,170 participants, 7.3% of men and 4.8% of women had positive impingement tests. Radiographic cam type findings were associated with positive tests in the men, but not women. A 2009 study from Asia reviewed records of 843 consecutive patients (978 hips) who underwent primary surgery for osteoarthritis or other diseases of the hip to determine the prevalence of FAI in this population. (5) Twenty-six patients (32 hips) were excluded due to insufficient radiographs or records, resulting in a study population of 817 patients (946 hips). The average age at the time of surgery was 54.8 years (range: 12-92 years). The majority of patients (73%) were diagnosed with osteoarthritis secondary to developmental dysplasia of the hip, another 12% had idiopathic osteonecrosis, and 1.7% had Legg-Calve-Perthes disease. Only 17 patients (1.8%) were considered to have had primary osteoarthritis. Of these, 6 patients (average age: 63 years; range: 32-79) were determined to have FAI from preoperative radiographs, resulting in a possible etiology of FAI for 0.6% of the total population undergoing surgery for osteoarthritis and 35% in the population with primary osteoarthritis.

Idiopathic Arthritis. Bardakos and Villar retrospectively examined progression of osteoarthritis of 43 patients (43 hips) younger than 55 years of age with a history of symptomatic idiopathic arthritis, first seen no later than 1997, who exhibited pistol-grip deformity of the femur and mild to moderate osteoarthritis (Tonnis grade 1 or 2) at baseline. (6) Radiographs taken at least 10 years apart showed progression of osteoarthritis in two thirds of the patients, with 12 receiving hip replacement or resurfacing after more than 10 years. Logistic regression analysis showed the medial proximal femoral angle and the posterior wall sign as the only significant independent predictors for progression of osteoarthritis in this small sample. A reduction of one degree in the medial proximal angle increased the odds of the osteoarthritis progressing by 21 times, while osteoarthritis in a hip with a positive posterior wall sign (the center of the femoral head located lateral to the outline of the of the posterior acetabular rim) was 10 times more likely to progress than a hip that had a negative posterior wall sign. Of note, one third of the patients with a pistol-grip deformity did not progress rapidly within the assessment period.

In another study, Tanzer and Noiseux reported that of 38 consecutive patients who were treated arthroscopically and who had a labral tear, 97% were found to have a pistol-grip deformity on preoperative radiographs. (7) These authors also reported that in 200 consecutive patients (200 hips) having primary total hip arthroplasty (THA), the underlying etiology of patients' arthritis was determined by their history and radiographic findings. All patients without a history or radiographic evidence of underlying hip disease were given the diagnosis of idiopathic hip arthritis. From the 125 cases diagnosed as idiopathic arthritis, 100% exhibited a pistol-grip deformity. Radiographs of the contralateral limb showed that 31% of patients had a healthy hip without a deformity or evidence of osteoarthritis, 14% had a deformity without evidence of arthritis, and 55% had a pistol-grip deformity and radiographic evidence of arthritis. A pistol-grip deformity was associated with arthritis later in life.

Kim et al. reviewed outcomes of 43 patients (mean age: 40 years; range: 18-68 years) who had labral tears and early osteoarthritis (Tonnis grade 0 to 1, average Japanese Orthopedic Association [JOA] scores of less than 1) and symptoms lasting 3 months or more who had been treated with debridement. (8) At an average 50 months' follow-up (12-96 months), 74% of patients had improved, with 11 showing no improvement. Blinded evaluation of preoperative radiographs and magnetic resonance (MR) arthrograms indicated that 42% of patients had FAI. When treated only with debridement, patients were less likely to improve if early stage osteoarthritis or FAI was present at the time of surgery. For example, on the JOA scale where 0=severe pain to 3=no pain, patients without either FAI or osteoarthritis scored 2.6 at follow-up, while patients with FAI scored 1.83, and those with both FAI and osteoarthritis scored 1.67.

Slipped Capital Femoral Epiphysis. Sink et al. reported a retrospective review from 2 U.S. centers on 36 patients (39 hips) with stable slipped capital femoral epiphysis (SCFE) who were treated with open surgical hip dislocation for chronic symptoms. (9) The degree of slip was considered to be mild in 8, moderate in 19, and severe in 11 patients, and the average time between in situ pinning and surgical hip dislocation was 20 months (range: 6-48 months). The majority of patients had partial or complete relief of symptoms immediately after initial pinning followed by a recurrence of symptoms that were consistent with impingement. All but one patient had either labral or cartilage injury, with labral injury observed in 34 of 39 hips and cartilage injury in 33 or 39 hips (5 grade 1, 10 grade 2, 4 grade 3, 10 grade 4, and 4 grade 5); the average depth of cartilage damage was 5 mm (range: 2-10 mm). There was no correlation between slip severity or duration of symptoms and the type of cartilage injury.

Dodds et al. examined the prevalence of FAI in 36 patients (49 hips) who returned for clinical evaluation at an average 6 years after SCFE. (10) There was no difference in the grade of slip between those patients who were available for follow-up and the total cohort treated for SCFE. The average age at presentation was 12.2 years, and at the time of evaluation all patients had reached skeletal maturity. Postoperative radiographs were reviewed for the grade of slip, Southwick slip angle, Loder's classification of physeal stability, and the anterior head-neck offset (alpha) angle. Pain and impingement were found in 30% of the 30 hips with grade 1 slips, 25% of the 8 hips with grade 2 slips, and 0% of the 4 hips with grade 3 slips. None of the radiographic factors including the grade of slip was predictive of subsequent impingement; the alpha angle was the most influential variable in regression analysis (p=0.63). Together, these results indicate that it is difficult to predict which patients with SCFE will develop FAI but that all children should be followed into adulthood and monitored for impingement.

Athletic Pubalgia. The incidence of athletic pubalgia symptoms in professional athletes with symptomatic FAI was reported by Hammoud et al. in 2012. (11) Out of 38 professional athletes who underwent arthroscopic treatment for FAI, 32% had previously undergone surgery for athletic pubalgia. While none of the patients had returned to their previous level of play after isolated athletic pubalgia surgery, all 12 patients with treatment of both athletic pubalgia and FAI returned to professional competition. In another subset of 15 patients who presented with both athletic pubalgia and FAI, both sets of symptoms resolved after treatment of FAI alone.

In summary, evidence on the natural history and long-term effect of treatment is limited due to the relatively recent recognition of this condition. Overall, the retrospective evidence available indicates a relatively strong association between cam-type impingement related to a pistol-grip deformity, labral damage, and the subsequent development of osteoarthritis. The identification of patients with FAI morphology who will progress to osteoarthritis (and perhaps more importantly those who are unlikely to progress) is limited at this time, although some evidence from retrospective studies is beginning to emerge. An association between FAI and athletic pubalgia is also emerging.

Treatment of FAI with Arthroscopic or Open Approaches

Bedi and colleagues performed a systematic review of the literature on labral tears and FAI, with a literature search conducted in May 2008. (12) Level I, II, III, or IV study designs were included if the patient population had a labral tear and/or FAI as the major diagnosis. Patients with severe pre-existing osteoarthritis or acetabular dysplasia were excluded. Of the 19 articles included, only 1 met the criteria for level III basis of evidence. The studies reviewed suggested that 65% to 85% of patients will be satisfied with their outcome at a mean of 40 months after surgery. All series reported an increased incidence of failure among patients with substantial pre-existing osteoarthritis. The authors concluded that the quality of literature reporting outcomes of surgical intervention for labral tears and FAI is limited. The larger case series, including those published after the literature search for the systematic review by Bedi et al, are described below.

Several systematic reviews comparing open and arthroscopic surgery for FAI have been identified. (13-15) Matsuda et al. included 18 level III or IV studies (controlled cohort or case series) with a minimum 1-year follow-up. (13) There were 6 papers on open surgical dislocation, 4 on mini-open procedures, and 8 arthroscopic studies. All 3 approaches were found to be effective in improving pain and function in short-term to midterm studies. Open dislocation surgery had a comparatively high major complication rate primarily because of trochanteric osteotomy-related issues. The mini-open method showed comparable efficacy but a significant incidence of iatrogenic injury to the lateral femoral cutaneous nerve. Botser and colleagues included 26 level II to IV articles totaling 1,462 hips in 1,409 patients. (14) Of these, 900 hips were treated arthroscopically, 304 with the open dislocation method, and 258 by the mini-open method. The mean time from onset of symptoms to surgery was 28 months. Overall complication rates were found to be 1.7% for the arthroscopic group, 9.2% for the open surgical dislocation group, and 16% for the combined approach group. For a 2012 review by Papalia et al., 31 studies (1,713 patients) were identified that reported clinical, functional, and imaging outcomes after open or arthroscopic treatment of FAI. (15)

A direct comparison of arthroscopic and open treatment of FAI was reported by Zingg et al. in 2013. (16) Out of 200 patients with FAI who were invited to participate in this prospective study, 10 patients agreed to be randomly allocated to arthroscopy or open surgical hip dislocation and 28 patients agreed to participate in the study but selected their preferred treatment. The open and arthroscopic groups were generally comparable at baseline. Arthroscopic treatment of FAI resulted in a shorter hospital stay (3 vs. 5 days) and less time off work. The Harris hip score (HHS) was improved compared to open treatment at 6 weeks, 3 months, and 12 months. Overall, pain scores (Western Ontario and McMaster Universities Arthritis Index [WOMAC] and Visual Analog Score [VAS]) were lower with arthroscopy, reaching statistical significance on about half of the time points. Compared with the open surgical approach, arthroscopy resulted in morphological over-corrections at the head-neck-junction.

Arthroscopic Approach

Previously, there were 4 prospective/consecutive case series with at least 100 patients/hips and 1 controlled cohort study. Literature updates have identified additional large series with longer follow-up.

The largest prospective series is by Malviya et al., who reported on changes in quality of life (QoL) for 612 patients who were treated by a single surgeon. (17) Patients ranged in age from 14 to 75 years (mean of 36.7). At 1 year after surgery, QoL scores on the Rosser Index improved by at least 1 grade in 76.6% of patients, were unchanged in 14.4%, and decreased in 9%. Byrd and Jones provided a brief report on 200 patients (207 hips) from a consecutive group of 220 patients (227 hips) who had been treated with arthroscopy for FAI. (18) The average age of the patients was 33 (range not reported), with symptoms averaging 32 months and no sign of advanced osteoarthritis. At an average of 16 months (range: 12-24 months) after treatment, patients showed an average 20-point improvement (-17 to 60) on the 91-point modified Harris Hip Score (MHHS). Eighty-three percent of patients were considered to be improved by the procedure.

In 2012, Palmer et al. reported prospective 3-year follow-up on 201 procedures for cam-type FAI with a Tonnis grade of 1 or less. (19) The mean duration of symptoms before surgery was 59 months. At follow-up, the Nonarthritic Hip Score (NAHS) improved from a mean of 56.1 to 78.2 and VAS for pain improved from 6.8 to 2.7. There was a higher incidence of grade 4 acetabular chondral defect in the 12 patients who required hip arthroplasty during the follow-up period compared with patients who did not undergo arthroplasty, and patients with pincer resection had poorer results (NAHS improvement of 16.1) compared to patients with only cam-type FAI (NAHS improvement of 23.9). Of the 93 patients who were able to return for a final postoperative radiograph, 91 (97.8%) had no change in the Tonnis grade. Subgroup analyses of patients who were 20 or younger and 60 or older showed no significant effect of age. Among the 48 patients who were excluded from this study due to acetabular chondral defects greater than 1.5 cm2, 60% underwent hip replacement at a mean of 21.7 months (range, 2-29 months).

Philippon et al. reported 2.3 year follow-up (range: 2-2.9 years) on 100 of 209 prospectively enrolled consecutive patients who underwent hip arthroscopy for disabling pain. (20) Patients were included in the study report (n=122) if they underwent arthroscopic treatment for FAI and chondrolabral dysfunction and did not have bilateral hip arthroscopy, avascular necrosis, or previous surgery. Of the 100 patients available for follow-up, 90 (90%) improved from an average score of 58 to 84 on the MHHS, and 10 (10%) required total hip arthroplasty (THA) at a mean of 16 months. Patients with a joint space of less than 2 mm were 39 times more likely to progress to THA. Larson and Giveans reported 10-month follow-up (3 months to 3 years) from a consecutive series of 96 patients (100 hips) who presented with FAI. (21) The average age was 35 (range: 16-64 years). Following FAI treatment, the impingement test was reported to be better in 86% of patients, with good to excellent results in 75% of patients. Three patients (3%) required THA, and 6 had heterotopic bone formation. VAS for pain improved from 6.7 at baseline to 1.9 at follow-up. Scores on the short-form (SF)-12 improved from 60 to 78.

Labral Repair. In 2013, Krych et al. reported a non-blinded RCT of labral repair versus labral debridement in 36 female patients with pincer-type or combined-type FAI. (22) At a mean 32-month follow-up (range 12 to 48 months), both groups showed significant improvement in the Hip Outcome Score (HOS) compared to baseline. Compared to the debridement group, the repair group had better outcomes on activities of daily living HOS (91.2 vs. 80.9) and sports HOS (88.7 vs. 76.3). A greater number of patients in the repair group rated their hip function as normal or nearly normal (94% vs. 78%).

Bardakos and colleagues compared results from 24 patients treated with osteochondroplasty for cam impingement (after 2004) with 47 patients who showed cam impingement but had only the labrum repaired (between 2000 and 2004). (23) The cohorts were matched for age (27-46 years) and for follow-up of 1 year. The number of patients who did not meet the selection criteria was not reported. There was a trend (p=0.11) for improved MHHS outcomes (excellent, good, fair, poor) in patients who were treated for impingement in addition to labral repair in this small study. Post hoc analysis of the percentage of patients in the excellent/good category showed significant improvement for the FAI-treated patients over historical controls (83% vs. 60%, respectively; p=0.043). Results of this study should be interpreted cautiously due to multiple potential sources of bias, including selection bias, limited follow-up, and the small sample size.

Revision Surgery. Two case series were identified on revision arthroscopic treatment of FAI. (24, 25) Philippon and colleagues reported on 37 revisions of previous hip arthroscopies by the senior author (51%) or referred from other centers. (24) Radiographic evaluation showed evidence of impingement in 36 of 37 patients that was either not addressed (60%) or inadequately addressed (32%) at the time of the index procedure. Five of the revisions (14%) required repeat revision or total hip replacement and were considered failures. Average 1-year follow-up on 27 of 32 hips that did not fail revision showed improvement (mean of 77; range: 36-100) on the MHHS. Heyworth et al. identified 24 revisions (23 patients) out of a total of 450 patients who underwent a hip arthroscopy at their institution. (25) The mean interval between the primary hip arthroscopy and recurrence of symptoms was 6 months (range: 0 to 39 months). Radiographic evaluation showed evidence of bony impingement in 19 cases (79%). Of these, 10 had only soft tissue repair during the primary procedure, and 9 had debridement of bone; 7 of the 9 were considered to be inadequate. Although the revision rate for arthroscopic FAI cannot be determined from the data provided, the authors commented that even when bony lesions are fully recognized, there may be a tendency to insufficiently address them surgically. Revision arthroscopy was also reported in 16 patients for the treatment of adhesions following open surgical hip dislocation for FAI. (26)

Poor outcomes following arthroscopic treatment of FAI in patients with arthritis have been reported. Larson et al. conducted a retrospective comparison of outcomes from arthroscopic treatment of 154 patients (169 hips) without joint space narrowing (Tonnis grade 0 to 1) and 56 patients (58 hips) with preoperative radiographic evidence of joint space narrowing (Tonnis grade 2 or 3). (27) Although both groups had improved scores throughout 12-month follow-up, outcomes were better for patients without osteoarthritis than for patients with osteoarthritis. Patients with advanced preoperative joint space narrowing (n=22) showed no improvement after treatment for FAI. At 3-year follow-up, the mean Harris Hip score was 88 for the group without osteoarthritis and 67 for the group with osteoarthritis. The failure rate at the last follow-up, defined as a MHHS less than 70 or conversion to hip arthroplasty, was 12% for patients without osteoarthritis, 33% for hips with mild to moderate preoperative joint space narrowing (<50% joint space narrowing or >2 mm joint space), and 82% failure rate for hips with advanced preoperative joint space narrowing (>50% joint space narrowing or < 2 mm joint space). Multiple linear regression analysis revealed that increasing radiographic joint space narrowing, chondral grade on magnetic resonance imaging (MRI), and greater duration of symptoms preoperatively were independent predictors for lower Harris Hip scores. Another study reported outcomes from 20 patients (out of a series of 150) who showed generalized severe cartilage lesions during intraoperative arthroscopic assessment for FAI. (28) Nine hips had Tonnis grade I osteoarthritis, 6 had grade II, and 5 had grade III osteoarthritis. At a mean follow-up of 3 years, 10 patients (50%) had undergone, or planned to undergo, total hip replacement. Preoperatively, 5 of the 10 hips had Tonnis grade III osteoarthritis. Another 2 patients had a poor result at latest follow-up but were not yet willing to undergo THA. The mean time between the index surgery and THA was 1.4 years (range, 0.4 to 2.2 years). The authors concluded that in patients with generalized chondral lesions, arthroscopic treatment of FAI does not have any effect beyond the short-term pain relief resulting from debridement.

Selection for Age. Philippon et al. evaluated outcomes following arthroscopic treatment of FAI in 153 consecutive patients aged 50 years or older. (29) The mean age of the patients was 57 years (range, 50 to 77 years). The prospective database included range of motion, MHHS, Hip Outcome Score (HOS) for activities of daily living, HOS for sports, and SF-12 score preoperatively and at 6-months after surgery. Questionnaires were then mailed annually. THA was required after arthroscopy for FAI in 20% of patients at a mean of 1.6 years (range, 3 months to 4 years). In the patients who did not require THA, the MHHS improved from 58 to 84, the HOS for activities of daily living improved from 66 to 87, and the HOS for sports improved from 42 to 72. The physical component of the SF-12 improved from 38 to 49, with no change in the mental component. Survivorship, defined as not requiring hip replacement, was 92% at 1 year, 84% at 2 years, and 80% at 3 years. For the 64 patients who had data available at 3 years, patients with greater than 2 mm of joint space preoperatively had survivorship of 90% whereas those with 2 mm or less of joint space had survivorship of 57%. Logistic regression modeling adjusted for age and days from injury to surgery identified joint space of 2 mm or less and preoperative MHHS of less than 50 as risk factors for hip replacement.

Javed and O'Donnell reported arthroscopic treatment of cam-type FAI in 40 patients older than 60 years of age (mean 65 years; range 60 to 82). (30) Patients were excluded from this retrospective study if they had Tonnis grade 2 or 3 osteoarthritis, pincer FAI, bilateral cam FAI, inflammatory or metabolic hip disease, hip dysplasia, Perthes disease, a history of fracture of the hip or previous surgery on the hip. Forty patients fulfilled the inclusion/exclusion criteria out of a total of 1,693 hip arthroscopies (2.4%) performed at their institution. In 17 patients there was no arthroscopic evidence of osteoarthritis in the hip; 23 had a variable degree of chondral loss from the acetabulum and/or femoral head. The MHHS and the non-arthritic hip score were collected pre-operatively and at 2, 6, 26, and 52 weeks post-operatively, and then on an annual basis. Follow-up was performed for a mean of 30 months (range, 12 to 54 months). The mean MHHS improved by 19.2 points (from 60.5 to 79.7), and the mean non-arthritic hip score improved by 15 points (from 62.1 to 77.2). Out of this selected group of 40 patients with unilateral cam impingement, equal to or less than Tonnis grade 1 osteoarthritis and a mean age of 63 years (range 60 to 70), 7 (17.5%), underwent total hip replacement at a mean interval of 12 months. All but one had evidence of severe synovitis, 4 of the 7 patients had grade 3 chondral loss from both the acetabulum and femoral head, while 3 had a grade 3 lesion of the acetabular cartilage. No fractures of the femoral neck occurred during the follow-up period.

Arthroscopic treatment for cam type FAI in adolescents with open growth plates was reported in 2012/2013. (31) At a mean follow-up of 14 months (range, 1-2 years), prospectively collected data showed improvement on the MHHS from 77.39 to 94.15 and on the nonarthritic hip score (NAHS) from 76.34 to 93.18. Of the 34 consecutive patients included in the study, 78.1% returned to full sporting activity. No complications (e.g., avascular necrosis, SCFE, fracture, or growth plate arrest) were observed.

Mixed Open/Arthroscopic Approach

A mixed open/arthroscopic approach for treatment of FAI was reported by Laude et al. in 97 patients (100 hips). (32) This technique allows direct visualization of the anterior femoral head-neck junction without dislocation. The average age of the patients was 33 (range: 16-56 years). Ninety-one (94%) were available for follow-up at an average 58 months (range: 29-104 months). Scores on the nonarthritic hip score (NAHS) increased from 55 at baseline to 84 at the last follow-up. One patient had a femoral neck fracture 3 weeks postoperatively, and 13 (14%) required revision due to persistent pain. In 8 of these patients, the damaged part of the labrum was removed, and in 6 patients, osteochondroplasty of the head was performed to improve the groove at the head-neck junction. Another patient had heterotopic ossification. Eleven hips (12%) required THA at a mean of 40 months (range: 5-75 months). In the THA group, the acetabular lesions were96respectively), and a higher percentage of Beck grade 5 was found (54% vs. 7%, respectively). The best results were observed in patients younger than 40 years with a Tonnis grade of 0.

Open Approach

Seven case series of patients with FAI treated with the open approach and dislocation were identified in the systematic review by Bedi et al. (12) Two studies reported on 5 patients, and 5 studies reported results from 19 to 52 patients, with follow-up ranging from 24 to 60 months. The 5 studies are briefly described here.

Beck et al. reported outcomes from 19 patients (average age: 36 years; range: 21-52) of 22 who had been selected from their database with confirmed clinical, radiographic, and MR arthrography diagnosis of FAI, had been treated with surgical dislocation of the hip and had at least 4 years of follow-up. (33) Three patients were excluded based on a history of prior intra-articular surgery of the involved hip. Of the remaining 19, all had labral damage, and 18 had acetabular damage. By 4 to 5 years' follow-up, 5 patients (26%) had undergone THA, with the failures associated with cartilage damage. Thirteen patients (68%) were reported to have had good to excellent outcomes.

Another study selected 52 of 141 consecutive patients to compare the effect of reattaching or removing the labrum during treatment for FAI. (34) Patients were selected for age (20-40 years) and no prior surgery; all had preoperative evidence of acetabular damage. Patients were excluded from the study because of incomplete clinical or radiographic documentation (n=48), open growth plates (n=4), age of greater than 40 years (n=29), previous hip surgery (n=7), or participation in professional athletic activity (n=1). Independent evaluations of 2-year follow-up indicated improved Merle d'Aubigne scores for both groups, from a baseline of 12 to 15 in the group in which the labrum was resected and from 12 to 17 in the group where the labrum was reattached. The study also found a reduction in progression to osteoarthritis if the labrum was reattached.

Peters and Erickson reported on 29 patients (30 hips) in a prospective protocol with minimum 2-years' follow-up. (35) The specific diagnoses were primary femoroacetabular impingement in 25 patients (26 hips), Legg-Calve-Perthes disease (n=3), and SCFE (n=1). The average age of the patients was 31 years (range: 16-51 years). Twenty-nine of the 30 hips had either cam-type impingement (n=14), or mixed cam and pincer-type impingement (n=15). Eighteen hips were reported to have had severe cartilage damage that was not seen on MR arthrography. The Harris Hip score improved from 70 at baseline to 87 at an average 32-months' follow-up. No progression to osteoarthritis was observed in 68% of patients. There was non-union in 8 hips (27%); 5 hips (17%) were expected to convert to THA due to progressive pain, and 4 (13%) had progression to osteoarthritis. Radiographic signs of progression of osteoarthritis and clinical failure requiring conversion to THA were seen only in patients with severe damage to the acetabular-articular cartilage. Two additional retrospective studies (n=23 and 34) that included patients with severe cartilage damage reported that 50% to 70% of patients improved and 30% to 50% failed (either no improvement or underwent subsequent THA) following open osteochondroplasty with dislocation. (36, 37)

Slipped Capital Femoral Epiphysis. Several articles have been identified from specialized centers on the treatment of symptomatic FAI in children with developmental hip disorders. The largest series on SCFE was a joint retrospective review from the Swiss group of Ganz and Leunig (n=30), together with the Children's Hospital of Boston (n=10), with 1- to 8-year follow-up on 40 patients (between 9 and 18 years of age) with moderate to severe SCFE who were treated by capital realignment with surgical dislocation. (38) The primary aim of the article was to determine whether this capital realignment technique was feasible and repeatable and would restore hip anatomy and function while avoiding osteonecrosis. Dislocation was not performed in SCFE with a slip angle of less than 30 degrees, in which trimming of the anterior metaphysis was considered sufficient to restore the anterior offset without weakening the femoral neck. No patients from either institution developed osteonecrosis, infection, deep venous thrombosis, or nerve palsies. Three patients developed delayed unions, none developed nonunions. Five patients required additional surgery for heterotopic ossification (n=1), residual impingement (n=1), or breakage of screw or wire fixation (n=3). The short-term postoperative clinical outcomes were found to be near normal, with similar scores between the operative and nonoperative hips. Stability and the duration of symptoms of SCFE (1 day to 3 years) were associated with the severity of acetabular cartilage damage observed at the time of surgery.

From the same U.S. institution was a 2006 report of 19 patients (12-43 years of age) who underwent either femoral neck osteoplasty (n=13) or osteoplasty with intertrochanteric osteotomy (n=6) via Ganz-type surgical dislocation. (39) Out of 12 patients with a history of SCFE (12-38 years of age), 9 were found to be improved at 8-25 months' follow-up. Out of the 7 patients (17-43 years of age) without SCFE who underwent open surgical dislocation for pistol-grip deformities, 5 had worse symptoms or minimal relief. Outcomes for patients with a chondral flap were worse than for patients without a chondral flap. For example, function scores on the Western Ontario and McMaster Universities Osteoarthritis Index (WOMAC) improved from a baseline of 26 to 10 in patients without a chondral flap but did not improve (25 to 24) in patients with chondral flap damage.

Summary

Five prospective/consecutive case series with more than 100 hips/patients treated for FAI have been identified. These studies show a 20-point improvement on the MHHS at short to mid-term follow-up, indicating a change from marked pain with a serious limitation of activities pre-operatively to mild pain after treatment, or from moderate pain with some limitations of ordinary activity or work to slight or no pain after treatment. Given that the arthroscopic procedure was developed around 2004, long-term follow-up is limited.

What can be ascertained from the current literature:

  • Not all patients with FAI morphology will have FAI pathology.
  • There is a high association between FAI pathology and idiopathic osteoarthritis, but this may represent a small proportion of the total cases of hip osteoarthritis.
  • Patients may present with hip pain that can be diagnosed as FAI by a combination of clinical evaluation, radiographs, and MR arthrography.
  • In cases in which there is a positive impingement test result, anterosuperior labral or acetabular damage identified on MR arthrography and a pistol-grip morphology identified on imaging, there is a very high probability that the acetabular damage is caused by impingement of the femoral head-neck junction against the acetabular rim. FAI can be verified intraoperatively.
  • Repair of the labrum alone can improve symptoms in the short term. It is reasonable to expect that debridement/osteoplasty of the bump or bone spur would reduce continued abrasion in the long term. Some studies, albeit of low quality, support this view.
  • Treatment of FAI is most effective in younger patients without osteoarthritis (Tonnis grade 0 or I) or severe cartilage damage. Although osteoarthritis can be identified with plain film radiographs, articular damage is not always identified with current imaging techniques.
  • There is a high probability that symptoms in patients with osteoarthritis (Tonnis grade II or III, or joint space of less than 2 mm) or severe cartilage damage (Outerbridge grade IV) will not improve following osteoplasty. These patients may require THA for progressing pain within 5 years.
  • In large case series, arthroscopic treatment of FAI in young to middle-age patients without osteoarthritis and showing mild to moderate cartilage damage results in 75% to 85% of patients improved.
  • Smaller case series suggest that open treatment of FAI in young to middle-age patients with moderate to severe cartilage damage results in 50% to 70% of patients improved. Non-union has been reported to occur in 27% of patients following the transection of the great trochanter with hip dislocation.

What cannot be ascertained from the literature:

  • It is not known which patients with FAI morphology are most likely to progress to osteoarthritis. The progression of pincer impingement with damage initially restricted to the labrum may follow a different time course than cam-type impingement.
  • It is not known whether treatment of FAI will reduce the occurrence of osteoarthritis.

Based on 1) the intraoperatively established relationship between FAI morphology and damage to the acetabulum, 2) the consistent improvement in symptoms reported in large prospective case series, and 3) the potential for continued and irreparable cartilage damage if FAI pathology is not addressed, it may be considered medically necessary to debride the bone at the same time that the labrum and/or articular cartilage is being repaired when specific criteria are met. Because of the differing benefits and risks of open and arthroscopic approaches, patients should make an informed choice.

The evidence is insufficient to permit conclusions concerning the effect of this procedure on the development of osteoarthritis. Therefore, treatment of FAI morphology in the absence of symptoms is considered investigational.

Due to the unclear balance of risks and benefits, questions regarding whether, when, and how to treat symptomatic FAI in children with SCFE are difficult. Although the impact of not treating FAI is established, there is limited evidence on treatment outcomes in pediatric patients. The open dislocation procedure is technically demanding with a high risk of serious complications and has not been shown to be safe and effective outside of a few highly specialized centers. In addition, questions remain concerning selection criteria and the appropriate timing and approach for FAI treatment in patients with developmental hip disorders. In a 2009 review of SCFE, surgeons from Children's Hospital Boston considered subcapital correction osteotomy with surgical dislocation to be an emerging treatment, stating that, "Currently, we recommend that this type of treatment should be restricted to few select specialized centers until the availability of long-term results and outcome. Also, this type of treatment has a steep learning curve, and it is advised to learn this surgical technique at a specialized center." (40) Since this approach has not been shown to be safe and effective outside of a few specialized centers, surgical treatment of FAI in pediatric patients is considered investigational.

Practice Guidelines and Position Statements

In 2011, the U.K.'s National Institute for Health and Clinical Excellence (NICE) issued revised guidance on arthroscopic femoroacetabular surgery for hip impingement syndrome. (41) NICE considers current evidence on the efficacy of arthroscopic femoroacetabular surgery for hip impingement syndrome to be adequate in terms of symptom relief in the short and medium term. There are well-recognized complications, and NICE recommends that this procedure may be used provided that normal arrangements are in place for clinical governance, consent and audit with local review of outcomes. Clinicians in the U.K. should submit details of all patients undergoing this procedure to a new register for arthroscopic femoroacetabular surgery for hip impingement syndrome from the British Hip Society to provide information about long-term outcomes. In addition, arthroscopic femoroacetabular surgery for hip impingement syndrome should only be carried out by surgeons with specialist expertise in arthroscopic hip surgery. This interventional procedure guidance (IPG 408) replaces IPG 213 from 2007.

Coding

Disclaimer for coding information on Medical Policies

Procedure and diagnosis codes on Medical Policy documents are included only as a general reference tool for each policy. They may not be all-inclusive.

The presence or absence of procedure, service, supply, device or diagnosis codes in a Medical Policy document has no relevance for determination of benefit coverage for members or reimbursement for providers. Only the written coverage position in a medical policy should be used for such determinations.

Benefit coverage determinations based on written Medical Policy coverage positions must include review of the member’s benefit contract or Summary Plan Description (SPD) for defined coverage vs. non-coverage, benefit exclusions, and benefit limitations such as dollar or duration caps. 

ICD-9 Codes

718.05, 718.75, 718.85, 719.45, 719.85

Procedural Codes: 29914, 29915, 29916, 27299
References
  1. Beck M, Kalhor M, Leunig M et al. Hip morphology influences the pattern of damage to the acetabular cartilage: femoroacetabular impingement as a cause of early osteoarthritis of the hip. J Bone Joint Surg Br 2005; 87(7):1012-8.
  2. Reichenbach S, Leunig M, Werlen S et al. Association between cam-type deformities and magnetic resonance imaging-detected structural hip damage: a cross-sectional study in young men. Arthritis Rheum 2011; 63(12):4023-30.
  3. Gosvig KK, Jacobsen S, Sonne-Holm S et al. Prevalence of malformations of the hip joint and their relationship to sex, groin pain, and risk of osteoarthritis: a population-based survey. J Bone Joint Surg Am 2010; 92(5):1162-9.
  4. Laborie LB, Lehmann TG, Engesaeter IO et al. Is a Positive Femoroacetabular Impingement Test a Common Finding in Healthy Young Adults? Clin Orthop Relat Res 2013 [Epub ahead of print].
  5. Takeyama A, Naito M, Shiramizu K et al. Prevalence of femoroacetabular impingement in Asian patients with osteoarthritis of the hip. Int Orthop 2009; 33(5):1229-32.
  6. Bardakos NV, Villar RN. Predictors of progression of osteoarthritis in femoroacetabular impingement: a radiological study with a minimum of ten years follow-up. J Bone Joint Surg Br 2009; 91(2):162-9.
  7. Tanzer M, Noiseux N. Osseous abnormalities and early osteoarthritis: the role of hip impingement. Clin Orthop Relat Res 2004; (429):170-7.
  8. Kim KC, Hwang DS, Lee CH et al. Influence of femoroacetabular impingement on results of hip arthroscopy in patients with early osteoarthritis. Clin Orthop Relat Res 2007; 456:128-32.
  9. Sink EL, Zaltz I, Heare T et al. Acetabular cartilage and labral damage observed during surgical hip dislocation for stable slipped capital femoral epiphysis. J Pediatr Orthop 2010; 30(1):26-30.
  10. Dodds MK, McCormack D, Mulhall KJ. Femoroacetabular impingement after slipped capital femoral epiphysis: does slip severity predict clinical symptoms? J Pediatr Orthop 2009; 29(6):535-9.
  11. Hammoud S, Bedi A, Magennis E et al. High incidence of athletic pubalgia symptoms in professional athletes with symptomatic femoroacetabular impingement. Arthroscopy 2012; 28(10):1388-95.
  12. Bedi A, Chen N, Robertson W et al. The management of labral tears and femoroacetabular impingement of the hip in the young, active patient. Arthroscopy 2008; 24(10):1135-45.
  13. Matsuda DK, Carlisle JC, Arthurs SC et al. Comparative systematic review of the open dislocation, mini-open, and arthroscopic surgeries for femoroacetabular impingement. Arthroscopy 2011; 27(2):252-69.
  14. Botser IB, Smith TW, Jr., Nasser R et al. Open surgical dislocation versus arthroscopy for femoroacetabular impingement: a comparison of clinical outcomes. Arthroscopy 2011; 27(2):270-8.
  15. Papalia R, Del Buono A, Franceschi F et al. Femoroacetabular impingement syndrome management: arthroscopy or open surgery? Int Orthop 2012; 36(5):903-14.
  16. Zingg PO, Ulbrich EJ, Buehler TC et al. Surgical hip dislocation versus hip arthroscopy for femoroacetabular impingement: clinical and morphological short-term results. Arch Orthop Trauma Surg 2013; 133(1):69-79.
  17. Malviya A, Stafford GH, Villar RN. Impact of arthroscopy of the hip for femoroacetabular impingement on quality of life at a mean follow-up of 3.2 years. J Bone Joint Surg Br 2012; 94(4):466-70.
  18. Byrd JW, Jones KS. Arthroscopic femoroplasty in the management of cam-type femoroacetabular impingement. Clin Orthop Relat Res 2009; 467(3):739-46.
  19. Palmer DH, Ganesh V, Comfort T et al. Midterm outcomes in patients with cam femoroacetabular impingement treated arthroscopically. Arthroscopy 2012; 28(11):1671-81.
  20. Philippon MJ, Briggs KK, Yen YM et al. Outcomes following hip arthroscopy for femoroacetabular impingement with associated chondrolabral dysfunction: minimum two-year follow-up. J Bone Joint Surg Br 2009; 91(1):16-23.
  21. Larson CM, Giveans MR. Arthroscopic management of femoroacetabular impingement: early outcomes measures. Arthroscopy 2008; 24(5):540-6.
  22. Krych AJ, Thompson M, Knutson Z et al. Arthroscopic labral repair versus selective labral debridement in female patients with femoroacetabular impingement: a prospective randomized study. Arthroscopy 2013; 29(1):46-53.
  23. Bardakos NV, Vasconcelos JC, Villar RN. Early outcome of hip arthroscopy for femoroacetabular impingement: the role of femoral osteoplasty in symptomatic improvement. J Bone Joint Surg Br 2008; 90(12):1570-5.
  24. Philippon MJ, Schenker ML, Briggs KK et al. Revision hip arthroscopy. Am J Sports Med 2007; 35(11):1918-21.
  25. Heyworth BE, Shindle MK, Voos JE et al. Radiologic and intraoperative findings in revision hip arthroscopy. Arthroscopy 2007; 23(12):1295-302.
  26. Krueger A, Leunig M, Siebenrock KA et al. Hip arthroscopy after previous surgical hip dislocation for femoroacetabular impingement. Arthroscopy 2007; 23(12):1285-89 e1.
  27. Larson CM, Giveans MR, Taylor M. Does arthroscopic FAI correction improve function with radiographic arthritis? Clin Orthop Relat Res 2011; 469(6):1667-76.
  28. Horisberger M, Brunner A, Herzog RF. Arthroscopic treatment of femoral acetabular impingement in patients with preoperative generalized degenerative changes. Arthroscopy 2010; 26(5):623-9.
  29. Philippon MJ, Schroder ESBG, Briggs KK. Hip arthroscopy for femoroacetabular impingement in patients aged 50 years or older. Arthroscopy 2012; 28(1):59-65.
  30. Javed A, O'Donnell JM. Arthroscopic femoral osteochondroplasty for cam femoroacetabular impingement in patients over 60 years of age. J Bone Joint Surg Br 2011; 93(3):326-31.
  31. Tran P, Pritchard M, O'Donnell J. Outcome of arthroscopic treatment for cam type femoroacetabular impingement in adolescents. ANZ J Surg 2012 [Epub ahead of print].
  32. Laude F, Sariali E, Nogier A. Femoroacetabular impingement treatment using arthroscopy and anterior approach. Clin Orthop Relat Res 2009; 467(3):747-52.
  33. Beck M, Leunig M, Parvizi J et al. Anterior femoroacetabular impingement: part II. Midterm results of surgical treatment. Clin Orthop Relat Res 2004; (418):67-73.
  34. Espinosa N, Rothenfluh DA, Beck M et al. Treatment of femoro-acetabular impingement: preliminary results of labral refixation. J Bone Joint Surg Am 2006; 88(5):925-35.
  35. Peters CL, Erickson JA. Treatment of femoro-acetabular impingement with surgical dislocation and debridement in young adults. J Bone Joint Surg Am 2006; 88(8):1735-41.
  36. Beaule PE, Le Duff MJ, Zaragoza E. Quality of life following femoral head-neck osteochondroplasty for femoroacetabular impingement. J Bone Joint Surg Am 2007; 89(4):773-9.
  37. Murphy S, Tannast M, Kim YJ et al. Debridement of the adult hip for femoroacetabular impingement: indications and preliminary clinical results. Clin Orthop Relat Res 2004; (429):178-81.
  38. Ziebarth K, Zilkens C, Spencer S et al. Capital realignment for moderate and severe SCFE using a modified Dunn procedure. Clin Orthop Relat Res 2009; 467(3):704-16.
  39. Spencer S, Millis MB, Kim YJ. Early results of treatment of hip impingement syndrome in slipped capital femoral epiphysis and pistol grip deformity of the femoral head-neck junction using the surgical dislocation technique. J Pediatr Orthop 2006; 26(3):281-5.
  40. Gholve PA, Cameron DB, Millis MB. Slipped capital femoral epiphysis update. Curr Opin Pediatr 2009; 21(1):39-45.
  41. National Institute for Health and Clinical Excellence (NICE). Arthroscopic femoro-acetabular surgery for hip impingement syndrome. IPG 408. 2011. Available at: www.nice.org.uk (Accessed March, 2012).
  42. Surgical Treatment of Femoroacetabular Impingement. Chicago, Illinois: Blue Cross Blue Shield Association Medical Policy Reference Manual (May 2013) Surgery 7.01.118.
History
Decedmber 2010  Policy updated with literature review through March 2010; references added and reordered; policy statement changed from "not medically necessary" to "investigational" if criteria for coverage not met.
September 2011  Policy updated with rationale and references, no change to policy statement. Codes 29914, 29915, 29916 added to policy; already on CRSP list since 1/2011
May 2012 CPT Codes 29914, 29915, 29916 added to the policy guidelines. no other changes made.
October 2013 Policy formatting and language revised.  Policy statement unchanged.  Title changed from "Femoroacetabular Impingement, Surgical Treatment" to "Femoroacetabular Impingement (FAI) Syndrome".  Removed CPT codes 27151, 29862, and 29999.
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Femoroacetabular Impingement, Surgical Treatment